Adinoff et al., “Vagal tone decreases following intravenous diazepam,” Psychiatry Research 41:89-97 (1992). |
Altschul et al., “Gapped BLAST and PSI-BLAST: a new generation of protein database search programs,” Nucleic Acids Res. 25:3389-3402 (1997). |
Anderson et al., “Direct interactins of coxsackievirus B3 with immune cells in the splenic compartment of mice susceptible or resistant to myocarditis,” J. Virol 70:4632-4645 (1996). |
Anzini et al., “Molecular basis of peripheral vs central benzodiazepine receptor selectivity in a new class of peripheral benzodiazepine receptor ligands related to alpidem,” J. Med. Chem. 4275 (1996). |
Arola et al., “Experimental Myocarditis induced by two different coxsackievirus B3 variants: aspects of pathogenesis and comparison of diagnostic methods,” J. Med. Virol. 47:251-259 (1995). |
Berkovich et al., “A natural processing product of rat diazepam binding inhibitor, triakontatetraneuropeptide (diazepam binding inhibitor 17-50) contains an ∀-helix, which allows discrimination between benzodiazepine binding site subtypes,” Mol. Pharmac. 37:164-172 (1990). |
Besman et al., “Identification of des-(Gly-Ile)-endozepine as an effector of corticotropin-dependent adrenal steroidogenesis: Stimulation of cholesterol delivery is mediated by the peripheral benzodiazepine receptor,” Proc. Natl. Acad. Sci. USA 86:4897-4901 (1989). |
Bishop et al., “Three abundance classes in HeLa cell messenger RNA,” Nature 250(463):199-204 (1974). |
Cappelli et al., “Mapping the peripheral benzodiazepine receptor binding site by conformationally restrained derivatives of 1-(2-Chlorophenyl)-N-methyl-N-(1-methylpropyl)-3-isoquinolinccarboxamide (PK11195),” J. Med. Chem. 2910 (1997. |
Charbonneau et al., “Peripheral-type benzodiazepine receptors in the living heart characterized by positron emission tomography,” Circulation 73:476-483 (1986). |
Chow et al., “Differential effects of myocarditic variants of coxsackievirus B3 in inbred mice, A pathologic characterization of heart tissue damage,” Lab. Invest. 64:55-64 (1991). |
Davies and Huston, “Peripheral benzodiazepine binding sites in heart and their interaction with dipyridamole,” Eur. J. Pharm. 73:209-211 (1981). |
DiMicco, “Evidence for control of cardiac vagal tone by benzodiazepine receptors,” Neuropharmacology 26:553-559 (1987). |
Edoute et al., “Ro 5-4864 and PK 11195, but not diazepam, depress cardiac function in an isolated working rat heart model,” Pharmacology 46:224-230 (1993). |
Garnier et al., “In vitro reconstitution of a functional peripheral-type benzodiazepine receptor from mouse leydig tumor cells,” Mol. Pharmac. 45:201-211 (1993). |
Grupp et al., “Benzodiazepine Ro 5-4864 increases coronary flow,” Eur. J. Pharm. 143:143-147 (1987). |
Guidotti., :Diazepam binding inhibtor (DBI): a peptide with multiple biological actions, Life Sci. 49(5):325-44 (1991). |
Hohenadl et al., “Strand-specific detection of enteroviral RNA in myocardial tissue by in situ hybridization,” Mol. Cell. Probes 5:11-20 (1991). |
Kruger et al., “Purification, Cloning, and Expression in a Peripheral-type Benzodiazepine Receptor,” in: GABA and Benzodiazepine Receptor Sybtypes, Biggio and Costa eds., pp. 1-14 (1990). |
Leeuwin et al., “Actions of enzodiazepines on the inotropy of the perfused rat heart,” Arch. Int. Pharmacodyn. 326:5-12 (1993). |
Leeuwin et al., “PK 11195 antagonizes the positive inotropic repsonse of the isolated rat heart to diazepam but not the negative inotropic response,” Eur. J. Pharm. 299:149-152 (1996). |
Leeuwin, R.S. et al., Modification of cardiac actions of RO 05-4864 by PK 11195 and flumazenil in the perfused rat heart. Life Sciences 61 (17):1631-42 (1997). |
McManus et al., “Direct myocardial injury by enterovirus: a central role in the evolution of murine myocarditis,” Clin. Immunol. Immunopathol. 68:159-169 (1993). |
Melnick et al., “Pathogenesis of coxsackie virus infection, Multiplication of virus and evolution of the muscle lesion in mice,” J. Expert. Med. 93:247-266 (1951). |
Mestre et al., “Electrophysiological and pharmacological characterization of peripheral benzodiazepine receptors in a guinea pig heart preparation,” Life Sciences 35:953-962 (1984). |
Needleman et al., “A general method applicable to the search for similarities in the amino acid sequence of two proteins,” J. Mol. Biol. 48:443 (1970). |
Parola et al., “Cloning and expression of a pharmacologically unique bovine peripheral-type benzodiazepine receptor isoquinoline binding protein,” J. Biol. Chem. 266:14,082-14,087 (1997). |
Pearson et al., “Improved tools for biological sequence comparison,” Proc. Natl. Acad Sci. USA 85:2444 (1988). |
Pfeffer et al., “Influence of Chronic Captopril Therapy on the Infacted Left Ventricle of the Rat,” Circ. Res. 57:84-95 (1985). |
Riond et al., “Molecular cloning and chroomosomal localizatin of a human periphreral-type benzodiazepine receptor,” Eur. J. Biochem. 195:305-311 (1991). |
Schoemaker et al., “Specific high-affinity binding sites for [3H]Ro 5-4864 in rat brain and kidney,” J. Pharmacol. Exp. Ther. 285:61-69 (1983). |
Shany et al., “Ro 5-4864 has a negative inotropic effect on human atrial muscle strips that is not antagonized by PK 11195,” Eur. J. Pharm. 253:231-236 (1994). |
Smith et al., “Comparison of biosequences,” Adv. Appl. Math. 2:482 (1981). |
Sprengel et al., “Molecular cloning and expression of cDNA encoding a peripheral-type benzodiazepine receptor,”J. Biol. Chem. 264:20,415-20,421 (1989). |
Taketani et al., “Involvement of peripheral-type benzodiazepine receptors in the intracellular transport of Heme and Porphyrins,” J. Biochem. 117:875-880 (1995). |