Dipiro et al. Pharmacotherapy A Pathophysiologic Approach. 1989 . pp. 960-961.* |
Arbiser et al. “Curcumin is an in vivo inhibitor of angiogenesis”; Molecular Medicine , vol. 4, No. 6, pp. 376-383; ISSN: 1076-1551, Jun. 1998.* |
Arbiser, et al., “Oncogenic H-ras stimulates tumor angiogenesis by two distinct pathways,” Proc Natl Acad Sci U S A. 94(3):861-6 (1997). |
Bille, et al., “Subchronic oral toxicity of turmeric oleoresin in pigs,” Food Chem Toxicol. 23(11):967-73 (1985). |
Conney, et al., “Inhibitory effect of curcumin and some related dietary compounds on tumor promotion and arachidonic acid metabolism in mouse skin,” Adv. Enzyme Regul. 31:385-96 (1991). |
Elayan, et al., “Long-term alteration in the central monoaminergic systems of the rat by 2,4,5-trihydroxyamphetamine but not by 2-hydroxy-4,5-methylenedioxymethamphetamine or 2-hydroxy-4,5-methylenedioxyamphetamine,” Eur. J. Pharmacol. 221 (2-3):281-8 (1992). |
Folkman, “Antiangiogenic gene therapy,” Proc. Natl. Acad. Sci. U. S. A. 95(16):9064-6 (1998). |
Folkman, “Diagnostic and therapeutic applications of angiogenesis research,” C. R. Acad. Sci. III. 316(9):914-18 (1993). |
Folkman, et al., “Long-term culture of capillary endothelial cells,” Proc. Natl. Acad. Sci. U. S. A. 76(10):5217-21 (1979). |
Genetic Engineering News, 18(17):1, 8, 34, 46 (1998). |
Golub, et al., “A non-antibacterial chemically-modified tetracycline inhibits mammalian collagenase activity,” J. Dent. Res. 1987 Aug;66(8):1310-4. |
Golub, et al., “Further evidence that tetracyclines inhibit collagenase activity in human crevicular fluid and from other mammalian sources,” J. Periodontal Res. 20(1):12-23 (1985). |
Golub, et al., “Minocycline reduces gingival collagenolytic activity during diabetes. Preliminary observations and a proposed new mechanism of action,” J. Periodontal Res. 18(5):516-26 (1983). |
Golub, et al., “Tetracyclines inhibit tissue collagenase activity. A new mechanism in the treatment of periodontal disease,” Periodontal Res. 19(6):651-5 (1984). |
Hong & Lippman, “Cancer chemoprevention,”J. Natl. Cancer Inst. Monogr. (17):49-53 (1995). |
Huang, et al., “Effect of dietary curcumin and ascorbyl palmitate on azoxymethanol-induced colonic epithelial cell proliferation and focal areas of dysplasia,” Cancer Lett. 64(2):117-21 (1992). |
Huang, et al., “Effects of curcumin, demethoxycurcumin, bisdemethoxycurcumin and tetrahydrocurcumin on 12-O-tetradecanoylphorbol-13-acetate-induced tumor promotion,” Carcinogenesis 16(10):2493-7 (1995). |
Huang, et al., “Suppression of c-Jun/AP-1 activation by an inhibitor of tumor promotion in mouse fibroblast cells,” Proc. Natl. Acad. Sci. U. S. A. 88(12):5292-6 (1991). |
Kelloff, et al., “Cancer Chemopreventive Agents: Drug Development Status II,” J. Cell Biochem. Suppl. 26:1-28 (1996). |
Kent, et al., “Requirement for protein kinase C activation in basic fibroblast growth factor-induced human endothelial cell proliferation,” Circ. Res. 77(2):231-8 (1995). |
Kenyon, et al., “A model of angiogenesis in the mouse cornea,” Invest. Ophthalmol. Vis. Sci. 37(8):1625-32 (1996). |
Kilpatrick, et al., “Towards an RNA-based therapy for Marfan syndrome,” Mol. Med. Today 4(9):376-81 (1998). |
Kohl, et al., “Inhibiton of farnesyltransferase induces regression of mammary and salivary carcinomas in ras transgenic mice,” Nat. Med. 1(8):792-7 (1995). |
Korutla, et al., “Inhibition of ligand-induced activation of epidermal growth factor receptor tyrosine phosphorylation by curcumin,” Carcinogenesis 16(8):1741-5 (1995). |
Larcher, et al., “Up-regulation of vascular endothelial growth factor/vascular permeability factor in mouse skin carcinogenesis correlates with malignant progression state and activated H-ras expression levels,” Cancer Res. 56(23):5391-6 (1996). |
Lu, et al., “Effect of curcumin on 12-O-tetradecanoylphorbol-13-acetate-and ultraviolet b light-induced expression of c-Jun and c-Fos in JB6 cells and in mouse epidermis,” Carcinogenesis 15(10):2363-70 (1994). |
Martell & Boothe, “The 6-deoxytetracyclines. VII. Alkylated aminotetracyclines possessing unique antibacterial activity,” J. Med. Chem. 10(1):44-6 (1967). |
O'Brien, et al. “Two mechanisms of basic fibroblast growth factor-induced angiogenesis in bladder cancer,” Cancer Res. 57(1):136-40 (1997). |
Prochaska, et al., “Rapid detection of inducers of enzymes that protect against carcinogens,” Proc. Natl. Acad. Sci. U. S. A. 89(6):2394-8 (1992). |
Rao, et al., “Chemoprevention of colon carcinogenesis by dietary curcumin, a naturally occurring plant phenolic compound,” Cancer Res. 55(2):259-66 (1995) |
Rao, et al., “Inhibition by dietary curcumin of azoxymethane-induced ornithine decarboxylase, tyrosine protein kinase, arachidonic acid metabolism and aberrant crypt foci formation in the rat colon,” Carcinogenesis 14(11):2219-25 (1993). |
Rao, et al., “Anti-inflammatory activity of curcumin analogues,” Indian J. Med. Res. 75:574-8 (1982). |