Adams, M.A. et al. “Vascular control mechanisms in penile erection: phylogeny and the inevitability of multiple and overlapping systems.” International Journal of Impotence Research, vol. 9, 1997, pp. 85-91. |
Adams, M.A., et al., “Enalapril can prevent vascular amplifier development in spontaneously hypertensive rats.” Journal of Hypertension, vol. 16, No. 3, pp. 252-260 (1990). |
Adams, M.A., et al., “Evidence in vivo for induction of cardiovascular growth processes by vasoconstrictor systems.” Blood Pressure, vol. 4, (Suppl 2), pp. 61-67 (1995). |
Adams, M.A., et al. “Common features regulating the systemic circulation and the penile vasculature,” Erectile Dysfunction. pp. 27-38. London: Martin Dunitz Ltd. 1998. |
Banting, J.D., et al., “Blunted cardiovascular growth induction during prolonged nitric oxide synthase blockade.” Hypertension, vol. 30, No. 3, Part 1, pp. 416-421 (1997). |
Childs, T.J., et al., “Regression of cardiac hypertrophy in spontaneously hypertensive rats by enalapril and the expression of contractile proteins,” Hypertension, vol. 16, No. 6, pp. 662-668 (1990). |
Christensen, K.L., et al., “Development of blood pressure in spontaneously hypertensive rats after withdrawal of long-term treatment related to vascular structure.” Hypertension, vol. 7, No. 2, pp. 83-90 (1989). |
Dukacz, S.A.W., et al., “The persistent effect of long-term enalapril on pressure natriuresis in spontaneously hypertensive rats.” American Journal of Physiology, vol. 273, pp. F104-F112 (1997). |
Friberg, P., et al., “Cardiac and vascular structural adaption in experimental hypertension.” European Heart Journal, vol. 11, (Supplement G), pp. 65-71 (1990). |
Gohike, P., et al., “Cardiac and vascular effects of chronic angiotensin converting enzyme inhibition at subantihypertensive doses.” Journal of Hypertension, vol. 10 (Supplement 6), pp. S141-S144 (1992). |
Harrap, S.B., et al., Proceedings of the HBPRCA. Transplantation studies of the role of the kidney in long-term blood pressure reduction following brief ACE Inhibitor treatment in young spontaneously hypertensive rats. Clinical and Experimental Pharmacology and Physiology, vol. 21, 129-131 (1994). |
Jespersen, L.T., et al., “Cardiac mass and peripheral vascular structure in hydralazine-treated spontaneously hypertensive rats.” Hypertension, vol. 7, No. 5, pp. 734-741 (1985). |
Kett, M.M., et al., “Enalapril does not prevent renal arterial hypertrophy in spontaneously hypertensive rats.” Hypertension, vol. 25, No. 3, pp. 335-342 (1995). |
Lee, R.M.K.W., et al., “Debate: vascular remodeling.” Journal of Hypertension, vol. 15, pp. 333-337 (1997). |
Lundle, M.J., et al., “Long-term inhibition of the renin-anglotensin system in genetic hypertension: analysis of the impact of blood pressure and cardiovascular structural changes,” Journal of Hypertension, vol. 15., No. 4, pp. 339-348 (1997). |
Moreau, P., et al., “Angiotensin II increases tissue endothelin and induces vascular hypertrophy. Reversal by ET,—receptor antagonist.” Circulation, vol. 96, No. 5, pp. 1593-1597 (1997). |
Morton, J.J., et al., “Angiotensin II receptor antagonist losartan has persistent effects on blood pressure in the young spontaneously hypertensive rat: lack of relation to vascular structure.” Journal of Vascular Research, vol. 29 pp. 264-269 (1992). |
Nyborg, N.C.B., et al., “Lack of effect of anti-hypertensive treatment with felodipine on cardiovascular structure of young spontaneously hypertensive rats.” Cardiovascular Research, vol. 19, pp. 528-536 (1985). |
Okabe, H., et al. “The penis is not protected—in hypertension there are vascular changes in the penis which are similar to those in other vascular beds.” International Journal of Impotence Research, vol. 11, 1999, pp. 133-140. |
Rizzoni, D., et al., “Effects of losartan and analapril on small artery structure in hypertensive rats.” Hypertension, vol. 32, pp. 305-310 (1998). |
Sano T., et al., “Differential structural responses of small resistance vessels to antihypertensive therapy.” Circulation, vol. 75, No. 3, pp. 618-626 (1987). |
Sharti, M., et al., “Remodeling of myocardium and arteries by chronic anglotensin converting enzyme inhibition in hypertensive patients.” Abstract, Journal of Hypertension, vol. 12, Suppl 4, pp. S37-S42 (1994). |
Thybo, N.K. et al., “Dose-dependent effects of perindopril on blood pressure and small-artery structure.” Hypertension, vol. 23, No. 5, pp. 659-666 (1994). |
Wahlander, H., et a., “Therapeutic, but not low-dose, angiotensin-converting enzyme inhibition causes regression of cardiovascular changes in spontaneously hypertensive rats.” Journal of Cardiovascular Pharmacology, vol. 27, pp. 327-334 (1996). |
Weiss, L., “Long-term treatment with antihypertensive drugs in spontaneously hypertensive rats (SHR). Effects on blood pressure, survival rate and cardiovascular design,” Acta. Physiol. Scand., vol. 91, pp. 393-408 (1974). |