The present invention relates generally to transistor body biasing circuits for integrated circuit devices, and more particularly to body biasing circuits for establishing body bias voltages upon power-up of such devices.
Integrated circuit (IC) devices can include transistors, such as insulated gate field effect transistors (hereinafter referred to as MOS type transistors, but not implying any particular gate or gate insulator material). MOS type transistors can include gates, drains, sources and bodies. In some IC devices, or some circuits within an IC device, the bodies of transistors have a permanent connection to a power supply voltage. For example, bodies of p-channel MOS transistors are connected to a high power supply voltage (e.g., VDD), while bodies of n-channel transistors are connected to a low power supply voltage (e.g., VSS).
However, in other IC devices or circuits within an IC device, the bodies of transistors can be biased to potential different from a power supply voltage. Such body biasing can advantageously alter the performance of the transistors. For example, a greater reverse body bias (i.e., p-channels body biased to a voltage above VDD or n-channel body biased to a voltage less than VSS) can decrease a transistor current leakage. Greater forward body bias (i.e., p-channels body biased to a voltage below VDD or n-channel body biased to a voltage above VSS) can increase transistor switching speed. In some cases, an IC device can include one or more body bias voltage generating circuits to generate desired body bias voltages, which are then applied to the bodies of target transistors.
A drawback to implementing body biasing circuits can be the need to strictly control body voltages during the transient state of powering up the IC device. If body voltages levels are not strictly controlled, a p-n junction formed by a transistor body can forward bias, drawing a large amount of current, and possibly damaging the IC device. Further, the initial floating states of transistor bodies can give rise to latch-up conditions (activation of a parasitic bipolar junction transistor).
Various embodiments of the present invention will now be described with reference to a number of drawings. The embodiments show circuits, integrated circuit (IC) devices, and methods for controlling body bias voltages during a power-up operation that can reduce current spikes and/or latch-up. Power-up operations can include those operations when a power supply voltage initially ramps up, including when a device is initially powered on, reset, subject to a power interruption event, or dynamically powered on and off for power conservation purposes, as but a few examples.
In the embodiments below, like items are referred to by the same reference character but with the leading digit(s) corresponding to the figure number.
Referring still to
As shown in
It is noted that the efficiency of such a body bias approach can be limited to:
where Vout is VBP or VBN. For example, assuming VBN=−0.6V and VDDIO=2.5 V, efficiency would be no greater than 24%.
In the embodiment shown, a body bias circuit 100 can receive a first power supply voltage (VDD) at a first power supply input 108-0, a second power supply voltage (VDDIO) at a second power supply input 108-1, and can generate a body bias voltage (VBx) at a body bias node 110. A body bias circuit 100 can include a body bias generator circuit 102, a switch circuit 104, and a voltage regulator 106. A voltage regulator 106 can receive VDDIO, and regulate it to achieve a predetermined level VDD_Reg (which can be the desired final level for VDD).
Switch circuit 104 can include a first switch input 112-0, second switch input 112-1, and switch output 112-2. First switch input 112-0 can receive the voltage VDD_Reg, generated from VDDIO. Second switch input 112-1 can receive voltage VDD. Switch circuit 104 can be controlled by a control signal CTRL generated by power-up circuits (not shown). On power up, CTRL can have a first value that results in first switch input 112-0 being connected to switch output 112-2, and second switch input 112-1 being isolated from switch output 112-2. CTRL can then change to a second value that results in first switch input 112-0 being isolated from switch output 112-2, and second switch input 112-1 being connected to switch output 112-2. In some embodiments, control signal CTRL can be based on VDD power-on reset (POR) type circuit, or the like.
Body bias generator circuit 102 can receive a power supply voltage VDDIn from the switch output 112-2. In response to VDDIn, body bias generator circuit 102 can generate a body bias voltage VBx. It is understood that VBx can be an n-channel body bias voltage or a p-channel body bias voltage.
Referring still to
It is understood that an IC device can include multiple body bias circuits like that shown in
While embodiments can include arrangements that switch a power supply voltage to body bias generator circuits, other embodiments can maintain (e.g., clamp) transistor bodies at a “safe” voltage upon power up. Subsequently, once body bias voltages are established, the transistor bodies can be switched from the clamped voltage to a desired body bias voltage. Examples of such embodiments will now be described.
In the embodiment shown, a body bias circuit 200 can utilize the first ramping power supply voltage (e.g., VDDIO) to enable clamping circuits to clamp body bias nodes to the other power supply voltages (e.g., VDD, VSS). Thus, as the device powers up, transistors bodies are clamped to such power supply voltages (e.g., VDD, VSS). Once the lower power supply voltage (VDD) is stable, body bias generator circuits can generate body bias voltages. The body bias nodes can then be “unclamped” from the power supply voltages and connected to the body bias voltages.
In the particular embodiment shown, a body bias circuit 200 can include a p-channel transistor body bias (PBB) generator circuit 202-0, an n-channel transistor body bias (NBB) generator circuit 202-1, a first clamp circuit 204-0, a second clamp circuit 204-1, and a clamp control circuit 214. PBB generator circuit 202-0 can generate a body bias voltage VBP_Gen for p-channel transistors. Similarly, NBB generator circuit 202-1 can generate a body bias voltage VBN_Gen for n-channel transistors. Both PBB and NBB generator circuits (202-0/1) can be powered with a first power supply voltage (VDD), which is understood to ramp up after a second power supply voltage (VDDIO). Thus, in an initial part of a power up operation, prior to VDD ramping up, PBB and NBB generator circuits (202-0/1) are not active, and thus are not generating their respective body bias voltages VBP_Gen, VBN_Gen.
Power supply voltage VDD can be provided from high power supply input 208-0 as a first input to first clamp circuit 204-0. First clamp circuit 204-0 can receive VBP_Gen from PBB generator circuit 202-0 as a second input. Output 210-0 of first clamp circuit 204-0 can set the body bias voltage VBP for some p-channel transistors at p-channel body bias node 210-0. First clamp circuit 204-0 can be controlled by a clamp enable signal (Clamp_EnableP) and a clamp disable signal (Clamp_DisableP). The clamp enable signal (Clamp_EnableP) can be activated from, or correspond to, VDDIO. Thus, an initial ramping of VDDIO can activate Clamp_EnableP. When Clamp_EnableP is active, clamp circuit 204-0 can clamp p-channel body bias node 210-0 to VDD. In contrast, the clamp disable signal (Clamp_DisableP) can be activated by clamp control circuit 214. When activated, signal Clamp_DisableP will override signal Clamp_EnableP, resulting in p-channel body bias node 210-0 being connected to VBP_Gen.
In a similar fashion to first clamp circuit 204-0, second clamp circuit 204-1 can receive VSS from a low power supply input 208-2, receive VBN_Gen from NBB generator circuit 202-1, and can set the body bias voltage VBN for some n-channel transistors at n-channel body bias node 210-1. Second clamp circuit 204-1 can operate in the same fashion as first clamp circuit 204-0. N-channel body bias node 210-1 can be clamped to VSS by operation of a VDDIO-based clamp enable signal (Clamp_EnableN). Such clamping can be disabled in response to a clamp disable signal (Clamp_DisableN), provided from clamp control circuit 214, resulting in n-channel body bias node 210-1 being connected to receive VBN_Gen.
Clamp control circuit 214 can operate according to VDD. That is, once VDD has reached a desired level, or some time afterward, clamp control circuit can activate the clamp disable signals (Clamp_DisableP, Clamp_DisableN).
One particular power up operation for the body bias circuit of
Referring to
At about time t1, VDDIO reaches a level that enables clamp circuits 204-0/1. As a result, VBP is clamped to VDD and VBN is clamped to VSS (which is zero volts in the embodiment shown).
At about time t2, VDD can ramp up. Due to clamping circuits 204-0/1 being enabled, VBP can remain clamped to VDD. Thus, VBP rises with VDD, preventing any forward biasing of p-channel body p-n junctions and/or potential latch-up conditions. Similarly, VBN remains clamped to VSS, also preventing the forward biasing of n-channel body junctions and/or reducing latch-up conditions. Once VDD reaches a sufficient level, body bias circuits (e.g., 202-0/1) can be activated, generating VBP and VBN voltages.
At about time t3, VDD reaches a desired level for a predetermined amount of time. As a result, clamp control circuit 214 can activate clamp disable signals, resulting in clamp circuits 204-0/1 connecting, or passively enabling the connection of, VBP to VBP_Gen and connecting, or passively enabling the connection of, VBN to VBN_Gen. The IC can now operate with transistors having the desired body bias voltage.
While
Generating body bias voltages (e.g., VBP, VBN) with VDD as opposed to VDDIO, as shown in embodiments herein, can be more efficient that conventional approaches. As noted above in connection with
A body bias circuit 300 can include items like those of
However, unlike the embodiment of
In very particular embodiments, clamp circuits 304-0/1 can utilize depletion mode MOS type transistors. Thus, in the absence of a gate voltage, such transistors can provide a clamping connection (i.e., the source-drain path connects VBN to VSS and/or VBP to VDD). Subsequently, a gate voltage can turn such depletion mode transistors off, enabling the VBN to be connected to VBN_Gen and VBP to be connected to VBP_Gen.
One particular power up operation for the body bias circuit of
Referring to
At about time t1, VDD reaches a desired level for a predetermined amount of time. As a result, clamp control circuit 314 can activate clamp disable signals, resulting in clamp circuits 304-0/1 connecting VBP to VBP_Gen and VBN to VBN_Gen. The IC can now operate with transistors having the desired body bias voltage.
As in other embodiments herein, body bias voltages (VBN_Gen, VBP_Gen) can be reverse body bias voltage, forward body bias voltages, as well as dynamically switching body bias voltages. In particular embodiments, a power supply voltage (VDD) and body bias voltages (VBP, VBN) can have the relationships and/or ranges as described for particular embodiments herein, and equivalents.
Current source circuit 418 can be connected between a gate of transistor N40 and a lower power supply connection 408-2. Current source circuit 418 can operate in response to a VDD level. That is, initially, when VDD is ramping up, current source circuit 418 may not draw current. However, once VDD reaches a predetermined level, or is stable for a predetermined amount of time, current source circuit 418 can draw current.
In operation, upon power up, VDD can start to ramp to a desired level. At this time, current source circuit 418 can be disabled. By operation of capacitance C40, a voltage at the gate of N40 can track its source (i.e., track VDD). Because N40 is a depletion mode device, it is conductive in this state, and p-channel body bias node 410-0 (i.e., VBP) can be clamped to VDD.
Subsequently, once VDD is at a suitable level, or stable for a suitable amount of time, current source circuit 418 can be enabled. As a result, the gate of N40 can be pulled toward VSS, turning off N40. In this way, p-channel body bias node 410-0 can be released from being clamped to VDD, and driven to body bias voltage, such as that generated by a body bias generator circuit (e.g., VBP_Gen).
In particular embodiments, a power supply voltage (VDD) and body bias voltage (VBP) can have the relationship and/or ranges as described for particular embodiments herein, and equivalents.
Capacitance C50 can be connected between the source and gate of transistor P50. Current source circuit 518 can be connected between a gate of transistor P50 and a high power supply (VDD) connection 508-0. Current source circuit 518 can operate in the same fashion as 418 shown in
Clamping device 516 can operate in a manner like that shown in
In particular embodiments, a power supply voltage (VSS) and body bias voltage (VBN) can have the relationship and/or ranges as described for particular embodiments herein, and equivalents.
Capacitance C60 can be connected between the source and gate of transistor N60. Current source circuit 618 can be connected between a gate of transistor N60 and a low power supply (VSS) connection 608-2. Current source circuit 618 can operate in the same fashion as 418 shown in
Clamping device 616 can operate in a manner like that shown in
In particular embodiments, a power supply voltage (VDD) and body bias voltage (VBP) can have the relationship and/or ranges as described for particular embodiments herein, and equivalents.
Capacitance C70 can be connected between the source and gate of JFET P70. Current source circuit 718 can be connected between a gate of JFET P70 and a high power supply (VDD) connection 708-0. Current source circuit 718 can operate in the same fashion as 418 shown in
Clamping device 716 can operate in a manner like that shown in
In particular embodiments, a power supply voltage (VSS) and body bias voltage (VBN) can have the relationship and/or ranges as described for particular embodiments herein, and equivalents.
It is understood that the clamp devices and transistors shown in
At time t0, VDD starts to ramp from 0 volts to 1.2 volts. Due to capacitive and resistive coupling VBP and VBN rise with VDD. Such a response can result in the forward biasing of p-n junctions and/or latch-up conditions.
At about time t1, VDD reaches a desired level of 1.2 V. VPB and VBN remain at elevated levels.
At about time t2, VPumpEN goes to an active level (high in this example). VBN and VBP can then be driven to desired body bias levels (various examples of possible body bias voltages are shown in
At time t0, VDDIO starts to ramp from 0 volts to 2.5 volts.
At time t1, after VDDIO reached 2.5 volts, VClampEN can be activated. As a result, VBP is clamped to VDD and VBN is clamped to VSS. After time t1, VDD can start to ramp from 0 volts to 1.2 volts. However, unlike
At time t2, VDD reaches its target value of 1.2 volts.
At time t3, after VDD has reached a stable value, VClampEN can return to an inactive level. Thus, VBP and VBN are released from being clamped to VDD and VSS, respectively.
At time t4, VPumpEN can be active, resulting in VBP and VBN being driven to desired body bias voltages.
In the particular embodiment shown, a clamping circuit 1000 includes a first clamping device 1016-0, a second clamping device 1016-1, and a control section 1040. A first clamping device 1016-0 can include an n-channel MOS type transistor N100 having a source and body connected to VDD, a drain connected to VBP, and a gate connected to receive VClampEN.
A control section 1040 can include a current mirror circuit formed by p-channel transistors P100/P101/P102, a resistor R100, and an n-channel MOS type transistor N101. Transistor P100 can have a source connected to a second power supply voltage VDDIO and a gate and drain connected to one another. VDDIO can ramp up before, and be greater than, VDD. Transistors P101/P102 can form a current mirror, having sources commonly connected to the gate-drain of P100. Transistor P101 can have its gate connected to its drain. Transistor P102 can have a gate connected to the gate of P101 and a drain connected to a control node 1042. Resistor R100 can be connected between the gate-drain of transistor P101 and the drain of transistor N101. Transistor N101 can have a gate that receives VClampEN and a body and source connected to VSS.
A control section 1040 can also include a disable circuit formed by n-channel MOS type transistors N102/N103/N104. Transistors N102 and N103 can have drains and gates commonly connected to control node 1042. Transistor N102 can have a source connected to the drain of transistor N104. Transistor N103 can have a source connected to n-channel body bias node VBN. The bodies of transistors N102/N104 can be commonly connected to each other. Transistor N104 can have a source connected to VSS.
A second clamping device 1016-1 can include an n-channel MOS type transistor N105 having a drain connected to VSS, a source connected to VBN, and a body connected to the body of N103.
In operation, VDDIO can ramp to a high level, enabling control section 1040.
Subsequently, VClampEN can be activated (e.g., driven to VDDIO). As a result, first clamping device 1016-0 can clamp VBP to VDD. In addition, VClampEN can turn on transistor N101, pulling one leg of the current mirror low. This results in control node 1042 being driven high. As a result, second clamping device 1016-1 can be turned on, clamping VBN to VSS.
VDD can then be ramped up, with VBP and VBN now clamped to VDD and VSS, respectively.
When VClampEN returns to an inactive level (e.g., VSS), first clamping device 1016-0 can turn off, isolating VBP from VDD. This can allow VBP to be driven to a desired body bias voltage. In addition, transistor N101 within the current mirror circuit turns off. As a result, control node 1042 can fall, turning off second clamping device 1016-1 to isolate VBN from VSS. This can allow VBN to be driven to a desired body bias level. Disable circuit N102/N103/N104 can ensure second clamping device 1016-1 remains turned off, even when VBN is driven below VSS.
While embodiments can be included in IC devices having conventional MOS type transistors, in some embodiments, all or a portion of the transistors within the integrated circuit device can be “deeply depleted channel” (DDC) transistors having an enhanced body effect as compared to conventional transistors. Accordingly, body bias voltages applied to DDC transistors can be used to modulate transistor responses more effectively than conventional MOS type transistors.
In some embodiments, transistors with driven body bias voltages can be DDC transistors. That is, bodies of DDC transistors can be clamped upon power-up as described herein, or equivalents. In addition or alternatively, the transistors making up body bias circuits can be DDC transistors. Thus, it is understood that any of the MOS type transistors described in the embodiments herein can be DDC transistors.
In
In some embodiments, a DDC transistor 1171, a highly doped screening region 1187 and optional threshold voltage set region 1189 can be made with dopants of the same conductivity type as the body (i.e., p-type dopant material in
Further descriptions of a DDC transistor as well as an exemplary fabrication process and other aspects of a DDC transistor can be found in U.S. Pat. No. 8,273,617, titled “Electronic Devices and Systems, and Methods for Making and Using the Same.”
As in the case of
It should be appreciated that in the foregoing descriptions of exemplary embodiments, various features are sometimes grouped together in a single embodiment, figure, or description thereof for the purpose of streamlining the disclosure aiding in the understanding of one or more of the various inventive aspects. This method of disclosure, however, is not to be interpreted as reflecting an intention that the claimed invention requires more features than are expressly recited in each claim. Rather, as the following claims reflect, inventive aspects lie in less than all features of a single foregoing disclosed embodiment. Thus, the claims following the detailed description are hereby expressly incorporated into this detailed description, with each claim standing on its own as a separate embodiment of the invention.
It is also understood that the embodiments may be practiced in the absence of an element and/or step not specifically disclosed. That is, an inventive feature of the invention may be elimination of an element.
Accordingly, while the various aspects of the particular embodiments set forth herein have been described in detail, the present invention could be subject to various changes, substitutions, and alterations without departing from the spirit and scope of the invention.
Number | Name | Date | Kind |
---|---|---|---|
3958266 | Athanas | May 1976 | A |
4000504 | Berger | Dec 1976 | A |
4021835 | Etoh et al. | May 1977 | A |
4242691 | Kotani et al. | Dec 1980 | A |
4276095 | Beilstein, Jr. et al. | Jun 1981 | A |
4315781 | Henderson | Feb 1982 | A |
4518926 | Swanson | May 1985 | A |
4559091 | Allen et al. | Dec 1985 | A |
4578128 | Mundt et al. | Mar 1986 | A |
4617066 | Vasudev | Oct 1986 | A |
4662061 | Malhi | May 1987 | A |
4761384 | Neppl et al. | Aug 1988 | A |
4780748 | Cunningham et al. | Oct 1988 | A |
4819043 | Yazawa et al. | Apr 1989 | A |
4885477 | Bird et al. | Dec 1989 | A |
4908681 | Nishida et al. | Mar 1990 | A |
4945254 | Robbins | Jul 1990 | A |
4956311 | Liou et al. | Sep 1990 | A |
4982113 | Jinbo | Jan 1991 | A |
5034337 | Mosher et al. | Jul 1991 | A |
5144378 | Hikosaka | Sep 1992 | A |
5156989 | Williams et al. | Oct 1992 | A |
5156990 | Mitchell | Oct 1992 | A |
5166765 | Lee et al. | Nov 1992 | A |
5208473 | Komori et al. | May 1993 | A |
5294821 | Iwamatsu | Mar 1994 | A |
5298763 | Shen et al. | Mar 1994 | A |
5369288 | Usuki | Nov 1994 | A |
5371419 | Sundby | Dec 1994 | A |
5373186 | Schubert et al. | Dec 1994 | A |
5384476 | Nishizawa et al. | Jan 1995 | A |
5426328 | Yilmaz et al. | Jun 1995 | A |
5444008 | Han et al. | Aug 1995 | A |
5552332 | Tseng et al. | Sep 1996 | A |
5559368 | Hu et al. | Sep 1996 | A |
5608253 | Liu et al. | Mar 1997 | A |
5622880 | Burr et al. | Apr 1997 | A |
5624863 | Helm et al. | Apr 1997 | A |
5625568 | Edwards et al. | Apr 1997 | A |
5641980 | Yamaguchi et al. | Jun 1997 | A |
5663583 | Matloubian et al. | Sep 1997 | A |
5712501 | Davies et al. | Jan 1998 | A |
5719422 | Burr et al. | Feb 1998 | A |
5726488 | Watanabe et al. | Mar 1998 | A |
5726562 | Mizuno | Mar 1998 | A |
5731626 | Eaglesham et al. | Mar 1998 | A |
5736419 | Naem | Apr 1998 | A |
5753555 | Hada | May 1998 | A |
5754826 | Gamal et al. | May 1998 | A |
5756365 | Kakumu | May 1998 | A |
5763921 | Okumura et al. | Jun 1998 | A |
5780899 | Hu et al. | Jul 1998 | A |
5847419 | Imai et al. | Dec 1998 | A |
5854561 | Arimoto et al. | Dec 1998 | A |
5856003 | Chiu | Jan 1999 | A |
5861334 | Rho | Jan 1999 | A |
5877049 | Liu et al. | Mar 1999 | A |
5885876 | Dennen | Mar 1999 | A |
5889315 | Farrenkopf et al. | Mar 1999 | A |
5895954 | Yasamura et al. | Apr 1999 | A |
5899714 | Farremkopf et al. | May 1999 | A |
5918129 | Fulford, Jr. et al. | Jun 1999 | A |
5923067 | Voldman | Jul 1999 | A |
5923987 | Burr | Jul 1999 | A |
5933047 | Zhu | Aug 1999 | A |
5936868 | Hall | Aug 1999 | A |
5942932 | Shen | Aug 1999 | A |
5946214 | Heavlin | Aug 1999 | A |
5985705 | Seliskar | Nov 1999 | A |
5989963 | Luning et al. | Nov 1999 | A |
5991221 | Ishikawa | Nov 1999 | A |
6001695 | Wu | Dec 1999 | A |
6020227 | Bulucea | Feb 2000 | A |
6043139 | Eaglesham et al. | Mar 2000 | A |
6060345 | Hause et al. | May 2000 | A |
6060364 | Maszara et al. | May 2000 | A |
6066533 | Yu | May 2000 | A |
6072217 | Burr | Jun 2000 | A |
6087210 | Sohn | Jul 2000 | A |
6087691 | Hamamoto | Jul 2000 | A |
6088518 | Hsu | Jul 2000 | A |
6091286 | Blauschild | Jul 2000 | A |
6096611 | Wu | Aug 2000 | A |
6103562 | Son et al. | Aug 2000 | A |
6121153 | Kikkawa | Sep 2000 | A |
6147383 | Kuroda | Nov 2000 | A |
6153920 | Gossmann et al. | Nov 2000 | A |
6157073 | Lehongres | Dec 2000 | A |
6175582 | Naito et al. | Jan 2001 | B1 |
6184112 | Maszara et al. | Feb 2001 | B1 |
6190979 | Radens et al. | Feb 2001 | B1 |
6194259 | Nayak et al. | Feb 2001 | B1 |
6198157 | Ishida et al. | Mar 2001 | B1 |
6218892 | Soumyanath et al. | Apr 2001 | B1 |
6218895 | De et al. | Apr 2001 | B1 |
6221724 | Yu et al. | Apr 2001 | B1 |
6229188 | Aoki et al. | May 2001 | B1 |
6232164 | Tsai et al. | May 2001 | B1 |
6235597 | Miles | May 2001 | B1 |
6245618 | An et al. | Jun 2001 | B1 |
6252452 | Hatori | Jun 2001 | B1 |
6268640 | Park et al. | Jul 2001 | B1 |
6271070 | Kotani et al. | Aug 2001 | B2 |
6271551 | Schmitz et al. | Aug 2001 | B1 |
6288429 | Iwata et al. | Sep 2001 | B1 |
6297132 | Zhang et al. | Oct 2001 | B1 |
6300177 | Sundaresan et al. | Oct 2001 | B1 |
6313489 | Letavic et al. | Nov 2001 | B1 |
6319799 | Ouyang et al. | Nov 2001 | B1 |
6320222 | Forbes et al. | Nov 2001 | B1 |
6323525 | Noguchi et al. | Nov 2001 | B1 |
6326666 | Bernstein et al. | Dec 2001 | B1 |
6335233 | Cho et al. | Jan 2002 | B1 |
6358806 | Puchner | Mar 2002 | B1 |
6380019 | Yu et al. | Apr 2002 | B1 |
6391752 | Colinge et al. | May 2002 | B1 |
6426260 | Hshieh | Jul 2002 | B1 |
6426279 | Huster et al. | Jul 2002 | B1 |
6432754 | Assaderaghi et al. | Aug 2002 | B1 |
6444550 | Hao et al. | Sep 2002 | B1 |
6444551 | Ku et al. | Sep 2002 | B1 |
6449749 | Stine | Sep 2002 | B1 |
6461920 | Shirahata | Oct 2002 | B1 |
6461928 | Rodder | Oct 2002 | B2 |
6472278 | Marshall et al. | Oct 2002 | B1 |
6473282 | Lin | Oct 2002 | B1 |
6482714 | Hieda et al. | Nov 2002 | B1 |
6489224 | Burr | Dec 2002 | B1 |
6492232 | Tang et al. | Dec 2002 | B1 |
6500739 | Wang et al. | Dec 2002 | B1 |
6503801 | Rouse et al. | Jan 2003 | B1 |
6503805 | Wang et al. | Jan 2003 | B2 |
6506640 | Ishida et al. | Jan 2003 | B1 |
6518623 | Oda et al. | Feb 2003 | B1 |
6521470 | Lin et al. | Feb 2003 | B1 |
6534373 | Yu | Mar 2003 | B1 |
6541328 | Whang et al. | Apr 2003 | B2 |
6541829 | Nishinohara et al. | Apr 2003 | B2 |
6548842 | Bulucea et al. | Apr 2003 | B1 |
6551885 | Yu | Apr 2003 | B1 |
6552377 | Yu | Apr 2003 | B1 |
6573129 | Hoke et al. | Jun 2003 | B2 |
6576535 | Drobny et al. | Jun 2003 | B2 |
6600200 | Lustig et al. | Jul 2003 | B1 |
6620671 | Wang et al. | Sep 2003 | B1 |
6624488 | Kim | Sep 2003 | B1 |
6627473 | Oikawa et al. | Sep 2003 | B1 |
6630710 | Augusto | Oct 2003 | B1 |
6660605 | Liu | Dec 2003 | B1 |
6662350 | Fried et al. | Dec 2003 | B2 |
6667200 | Sohn et al. | Dec 2003 | B2 |
6670260 | Yu et al. | Dec 2003 | B1 |
6693333 | Yu | Feb 2004 | B1 |
6730568 | Sohn | May 2004 | B2 |
6737724 | Hieda et al. | May 2004 | B2 |
6743291 | Ang et al. | Jun 2004 | B2 |
6743684 | Liu | Jun 2004 | B2 |
6751519 | Satya et al. | Jun 2004 | B1 |
6753230 | Sohn et al. | Jun 2004 | B2 |
6760900 | Rategh et al. | Jul 2004 | B2 |
6770944 | Nishinohara et al. | Aug 2004 | B2 |
6787424 | Yu | Sep 2004 | B1 |
6797553 | Adkisson et al. | Sep 2004 | B2 |
6797602 | Kluth et al. | Sep 2004 | B1 |
6797994 | Hoke et al. | Sep 2004 | B1 |
6808004 | Kamm et al. | Oct 2004 | B2 |
6808994 | Wang | Oct 2004 | B1 |
6813750 | Usami et al. | Nov 2004 | B2 |
6821825 | Todd et al. | Nov 2004 | B2 |
6821852 | Rhodes | Nov 2004 | B2 |
6822297 | Nandakumar et al. | Nov 2004 | B2 |
6831292 | Currie et al. | Dec 2004 | B2 |
6835639 | Rotondaro et al. | Dec 2004 | B2 |
6852602 | Kanzawa et al. | Feb 2005 | B2 |
6852603 | Chakravarthi et al. | Feb 2005 | B2 |
6881641 | Wieczorek et al. | Apr 2005 | B2 |
6881987 | Sohn | Apr 2005 | B2 |
6891439 | Jachne et al. | May 2005 | B2 |
6893947 | Martinez et al. | May 2005 | B2 |
6900519 | Cantell et al. | May 2005 | B2 |
6901564 | Stine et al. | May 2005 | B2 |
6916698 | Mocuta et al. | Jul 2005 | B2 |
6917237 | Tschanz et al. | Jul 2005 | B1 |
6927463 | Iwata et al. | Aug 2005 | B2 |
6928128 | Sidiropoulos | Aug 2005 | B1 |
6930007 | Bu et al. | Aug 2005 | B2 |
6930360 | Yamauchi et al. | Aug 2005 | B2 |
6957163 | Ando | Oct 2005 | B2 |
6963090 | Passlack et al. | Nov 2005 | B2 |
6995397 | Yamashita et al. | Feb 2006 | B2 |
7002214 | Boyd et al. | Feb 2006 | B1 |
7008836 | Algotsson et al. | Mar 2006 | B2 |
7013359 | Li | Mar 2006 | B1 |
7015546 | Herr et al. | Mar 2006 | B2 |
7015741 | Tschanz et al. | Mar 2006 | B2 |
7022559 | Barnak et al. | Apr 2006 | B2 |
7036098 | Eleyan et al. | Apr 2006 | B2 |
7038258 | Liu et al. | May 2006 | B2 |
7039881 | Regan | May 2006 | B2 |
7045456 | Murto et al. | May 2006 | B2 |
7057216 | Ouyang et al. | Jun 2006 | B2 |
7061058 | Chakravarthi et al. | Jun 2006 | B2 |
7064039 | Liu | Jun 2006 | B2 |
7064399 | Babcock et al. | Jun 2006 | B2 |
7071103 | Chan et al. | Jul 2006 | B2 |
7078325 | Curello et al. | Jul 2006 | B2 |
7078776 | Nishinohara et al. | Jul 2006 | B2 |
7089513 | Bard et al. | Aug 2006 | B2 |
7089515 | Hanafi et al. | Aug 2006 | B2 |
7091093 | Noda et al. | Aug 2006 | B1 |
7105399 | Dakshina-Murthy et al. | Sep 2006 | B1 |
7109099 | Tan et al. | Sep 2006 | B2 |
7119381 | Passlack | Oct 2006 | B2 |
7122411 | Mouli | Oct 2006 | B2 |
7127687 | Signore | Oct 2006 | B1 |
7132323 | Haensch et al. | Nov 2006 | B2 |
7169675 | Tan et al. | Jan 2007 | B2 |
7170120 | Datta et al. | Jan 2007 | B2 |
7176137 | Perng et al. | Feb 2007 | B2 |
7186598 | Yamauchi et al. | Mar 2007 | B2 |
7189627 | Wu et al. | Mar 2007 | B2 |
7199430 | Babcock et al. | Apr 2007 | B2 |
7202517 | Dixit et al. | Apr 2007 | B2 |
7208354 | Bauer | Apr 2007 | B2 |
7211871 | Cho | May 2007 | B2 |
7221021 | Wu et al. | May 2007 | B2 |
7223646 | Miyashita et al. | May 2007 | B2 |
7226833 | White et al. | Jun 2007 | B2 |
7226843 | Weber et al. | Jun 2007 | B2 |
7230680 | Fujisawa et al. | Jun 2007 | B2 |
7235822 | Li | Jun 2007 | B2 |
7247896 | Oh et al. | Jul 2007 | B2 |
7256639 | Koniaris et al. | Aug 2007 | B1 |
7259428 | Inaba | Aug 2007 | B2 |
7260562 | Czajkowski et al. | Aug 2007 | B2 |
7294877 | Rueckes et al. | Nov 2007 | B2 |
7297994 | Wieczorek et al. | Nov 2007 | B2 |
7301208 | Handa et al. | Nov 2007 | B2 |
7304350 | Misaki | Dec 2007 | B2 |
7307471 | Gammie et al. | Dec 2007 | B2 |
7312500 | Miyashita et al. | Dec 2007 | B2 |
7323754 | Ema et al. | Jan 2008 | B2 |
7332439 | Lindert et al. | Feb 2008 | B2 |
7348629 | Chu et al. | Mar 2008 | B2 |
7354833 | Liaw | Apr 2008 | B2 |
7380225 | Joshi et al. | May 2008 | B2 |
7398497 | Sato et al. | Jul 2008 | B2 |
7402207 | Besser et al. | Jul 2008 | B1 |
7402872 | Murthy et al. | Jul 2008 | B2 |
7416605 | Zollner et al. | Aug 2008 | B2 |
7427788 | Li et al. | Sep 2008 | B2 |
7442971 | Wirbeleit et al. | Oct 2008 | B2 |
7449733 | Inaba et al. | Nov 2008 | B2 |
7462908 | Bol et al. | Dec 2008 | B2 |
7469164 | Du-Nour | Dec 2008 | B2 |
7470593 | Rouh et al. | Dec 2008 | B2 |
7485536 | Jin et al. | Feb 2009 | B2 |
7487474 | Ciplickas et al. | Feb 2009 | B2 |
7491988 | Tolchinsky et al. | Feb 2009 | B2 |
7494861 | Chu et al. | Feb 2009 | B2 |
7496862 | Chang et al. | Feb 2009 | B2 |
7496867 | Turner et al. | Feb 2009 | B2 |
7498637 | Yamaoka et al. | Mar 2009 | B2 |
7501324 | Babcock et al. | Mar 2009 | B2 |
7503020 | Allen et al. | Mar 2009 | B2 |
7507999 | Kusumoto et al. | Mar 2009 | B2 |
7510932 | Oh et al. | Mar 2009 | B2 |
7514766 | Yoshida | Apr 2009 | B2 |
7521323 | Surdeanu et al. | Apr 2009 | B2 |
7521988 | Shin | Apr 2009 | B2 |
7531393 | Doyle et al. | May 2009 | B2 |
7531836 | Liu et al. | May 2009 | B2 |
7538364 | Twynam | May 2009 | B2 |
7538412 | Schulze et al. | May 2009 | B2 |
7562233 | Sheng et al. | Jul 2009 | B1 |
7564105 | Chi et al. | Jul 2009 | B2 |
7566600 | Mouli | Jul 2009 | B2 |
7569456 | Ko et al. | Aug 2009 | B2 |
7586322 | Xu et al. | Sep 2009 | B1 |
7592241 | Takao | Sep 2009 | B2 |
7595243 | Bulucea et al. | Sep 2009 | B1 |
7598142 | Ranade et al. | Oct 2009 | B2 |
7605041 | Ema et al. | Oct 2009 | B2 |
7605060 | Meunier-Beillard et al. | Oct 2009 | B2 |
7605429 | Bernstein et al. | Oct 2009 | B2 |
7608496 | Chu | Oct 2009 | B2 |
7615802 | Elpelt et al. | Nov 2009 | B2 |
7622341 | Chudzik et al. | Nov 2009 | B2 |
7638380 | Pearce | Dec 2009 | B2 |
7639041 | Perisetty | Dec 2009 | B1 |
7642140 | Bae et al. | Jan 2010 | B2 |
7644377 | Saxe et al. | Jan 2010 | B1 |
7645665 | Kubo et al. | Jan 2010 | B2 |
7651920 | Siprak | Jan 2010 | B2 |
7655523 | Babcock et al. | Feb 2010 | B2 |
7673273 | Madurawae et al. | Mar 2010 | B2 |
7675126 | Cho | Mar 2010 | B2 |
7675317 | Perisetty | Mar 2010 | B2 |
7678638 | Chu et al. | Mar 2010 | B2 |
7681628 | Joshi et al. | Mar 2010 | B2 |
7682887 | Dokumaci et al. | Mar 2010 | B2 |
7683442 | Burr et al. | Mar 2010 | B1 |
7696000 | Liu et al. | Apr 2010 | B2 |
7704822 | Jeong | Apr 2010 | B2 |
7704844 | Zhu et al. | Apr 2010 | B2 |
7709828 | Braithwaite et al. | May 2010 | B2 |
7723750 | Zhu et al. | May 2010 | B2 |
7737472 | Kondo et al. | Jun 2010 | B2 |
7741138 | Cho | Jun 2010 | B2 |
7741200 | Cho et al. | Jun 2010 | B2 |
7745270 | Shah et al. | Jun 2010 | B2 |
7750374 | Capasso et al. | Jul 2010 | B2 |
7750381 | Hokazono et al. | Jul 2010 | B2 |
7750405 | Nowak | Jul 2010 | B2 |
7750682 | Bernstein et al. | Jul 2010 | B2 |
7755144 | Li et al. | Jul 2010 | B2 |
7755146 | Helm et al. | Jul 2010 | B2 |
7759206 | Luo et al. | Jul 2010 | B2 |
7759714 | Itoh et al. | Jul 2010 | B2 |
7761820 | Berger et al. | Jul 2010 | B2 |
7795677 | Bangsaruntip et al. | Sep 2010 | B2 |
7808045 | Kawahara et al. | Oct 2010 | B2 |
7808410 | Kim et al. | Oct 2010 | B2 |
7811873 | Mochizuki | Oct 2010 | B2 |
7811881 | Cheng et al. | Oct 2010 | B2 |
7818702 | Mandelman et al. | Oct 2010 | B2 |
7821066 | Lebby et al. | Oct 2010 | B2 |
7829402 | Matocha et al. | Nov 2010 | B2 |
7831873 | Trimberger et al. | Nov 2010 | B1 |
7846822 | Seebauer et al. | Dec 2010 | B2 |
7855118 | Hoentschel et al. | Dec 2010 | B2 |
7859013 | Chen et al. | Dec 2010 | B2 |
7863163 | Bauer | Jan 2011 | B2 |
7867835 | Lee et al. | Jan 2011 | B2 |
7883977 | Babcock et al. | Feb 2011 | B2 |
7888205 | Herner et al. | Feb 2011 | B2 |
7888747 | Hokazono | Feb 2011 | B2 |
7895546 | Lahner et al. | Feb 2011 | B2 |
7897495 | Ye et al. | Mar 2011 | B2 |
7906413 | Cardone et al. | Mar 2011 | B2 |
7906813 | Kato | Mar 2011 | B2 |
7910419 | Fenouillet-Beranger et al. | Mar 2011 | B2 |
7919791 | Flynn et al. | Apr 2011 | B2 |
7926018 | Moroz et al. | Apr 2011 | B2 |
7935984 | Nakano | May 2011 | B2 |
7941776 | Majumder et al. | May 2011 | B2 |
7945800 | Gomm et al. | May 2011 | B2 |
7948008 | Liu et al. | May 2011 | B2 |
7952147 | Ueno et al. | May 2011 | B2 |
7960232 | King et al. | Jun 2011 | B2 |
7960238 | Kohli et al. | Jun 2011 | B2 |
7968400 | Cai | Jun 2011 | B2 |
7968411 | Williford | Jun 2011 | B2 |
7968440 | Seebauer | Jun 2011 | B2 |
7968459 | Bedell et al. | Jun 2011 | B2 |
7989900 | Haensch et al. | Aug 2011 | B2 |
7994573 | Pan | Aug 2011 | B2 |
8004024 | Furukawa et al. | Aug 2011 | B2 |
8012827 | Yu et al. | Sep 2011 | B2 |
8029620 | Kim et al. | Oct 2011 | B2 |
8039332 | Bernard et al. | Oct 2011 | B2 |
8046598 | Lee | Oct 2011 | B2 |
8048791 | Hargrove et al. | Nov 2011 | B2 |
8048810 | Tsai et al. | Nov 2011 | B2 |
8051340 | Cranford, Jr. et al. | Nov 2011 | B2 |
8053340 | Colombeau et al. | Nov 2011 | B2 |
8063466 | Kurita | Nov 2011 | B2 |
8067279 | Sadra et al. | Nov 2011 | B2 |
8067280 | Wang et al. | Nov 2011 | B2 |
8067302 | Li | Nov 2011 | B2 |
8076719 | Zeng et al. | Dec 2011 | B2 |
8097529 | Krull et al. | Jan 2012 | B2 |
8103983 | Agarwal et al. | Jan 2012 | B2 |
8105891 | Yeh et al. | Jan 2012 | B2 |
8106424 | Schruefer | Jan 2012 | B2 |
8106481 | Rao | Jan 2012 | B2 |
8110487 | Griebenow et al. | Feb 2012 | B2 |
8114761 | Mandrekar et al. | Feb 2012 | B2 |
8119482 | Bhalla et al. | Feb 2012 | B2 |
8120069 | Hynecek | Feb 2012 | B2 |
8129246 | Babcock et al. | Mar 2012 | B2 |
8129797 | Chen et al. | Mar 2012 | B2 |
8134159 | Hokazono | Mar 2012 | B2 |
8143120 | Kerr et al. | Mar 2012 | B2 |
8143124 | Challa et al. | Mar 2012 | B2 |
8143678 | Kim et al. | Mar 2012 | B2 |
8148774 | Mori et al. | Apr 2012 | B2 |
8163619 | Yang et al. | Apr 2012 | B2 |
8164378 | Pietri | Apr 2012 | B2 |
8169002 | Chang et al. | May 2012 | B2 |
8170857 | Joshi et al. | May 2012 | B2 |
8173499 | Chung et al. | May 2012 | B2 |
8173502 | Yan et al. | May 2012 | B2 |
8176461 | Trimberger | May 2012 | B1 |
8178430 | Kim et al. | May 2012 | B2 |
8179530 | Levy et al. | May 2012 | B2 |
8183096 | Wirbeleit | May 2012 | B2 |
8183107 | Mathur et al. | May 2012 | B2 |
8185865 | Gupta et al. | May 2012 | B2 |
8187959 | Pawlak et al. | May 2012 | B2 |
8188542 | Yoo et al. | May 2012 | B2 |
8196545 | Kurosawa | Jun 2012 | B2 |
8201122 | Dewey, III et al. | Jun 2012 | B2 |
8214190 | Joshi et al. | Jul 2012 | B2 |
8217423 | Liu et al. | Jul 2012 | B2 |
8225255 | Ouyang et al. | Jul 2012 | B2 |
8227307 | Chen et al. | Jul 2012 | B2 |
8236661 | Dennard et al. | Aug 2012 | B2 |
8239803 | Kobayashi | Aug 2012 | B2 |
8247300 | Babcock et al. | Aug 2012 | B2 |
8255843 | Chen et al. | Aug 2012 | B2 |
8258026 | Bulucea | Sep 2012 | B2 |
8266567 | El Yahyaoui et al. | Sep 2012 | B2 |
8286180 | Foo | Oct 2012 | B2 |
8288798 | Passlack | Oct 2012 | B2 |
8299562 | Li et al. | Oct 2012 | B2 |
8324059 | Guo et al. | Dec 2012 | B2 |
20010014495 | Yu | Aug 2001 | A1 |
20020033511 | Babcock et al. | Mar 2002 | A1 |
20020042184 | Nandakumar et al. | Apr 2002 | A1 |
20020089370 | Shin | Jul 2002 | A1 |
20030006415 | Yokogawa et al. | Jan 2003 | A1 |
20030047763 | Hieda et al. | Mar 2003 | A1 |
20030122203 | Nishinohara et al. | Jul 2003 | A1 |
20030173626 | Burr | Sep 2003 | A1 |
20030183856 | Wiecczorek et al. | Oct 2003 | A1 |
20030215992 | Sohn et al. | Nov 2003 | A1 |
20040053457 | Sohn | Mar 2004 | A1 |
20040075118 | Heinemann et al. | Apr 2004 | A1 |
20040075143 | Bae et al. | Apr 2004 | A1 |
20040084731 | Matsuda et al. | May 2004 | A1 |
20040087090 | Grudowski et al. | May 2004 | A1 |
20040126947 | Sohn | Jul 2004 | A1 |
20040175893 | Vatus et al. | Sep 2004 | A1 |
20040180488 | Lee | Sep 2004 | A1 |
20050056877 | Rueckes et al. | Mar 2005 | A1 |
20050106824 | Alberto et al. | May 2005 | A1 |
20050116282 | Pattanayak et al. | Jun 2005 | A1 |
20050250289 | Babcock et al. | Nov 2005 | A1 |
20050280075 | Ema et al. | Dec 2005 | A1 |
20060017100 | Bol et al. | Jan 2006 | A1 |
20060022270 | Boyd et al. | Feb 2006 | A1 |
20060049464 | Rao | Mar 2006 | A1 |
20060068555 | Zhu et al. | Mar 2006 | A1 |
20060068586 | Pain | Mar 2006 | A1 |
20060071278 | Takao | Apr 2006 | A1 |
20060091481 | Li et al. | May 2006 | A1 |
20060154428 | Dokumaci | Jul 2006 | A1 |
20060157794 | Doyle et al. | Jul 2006 | A1 |
20060197158 | Babcock et al. | Sep 2006 | A1 |
20060203581 | Joshi et al. | Sep 2006 | A1 |
20060220114 | Miyashita et al. | Oct 2006 | A1 |
20060223248 | Venugopal et al. | Oct 2006 | A1 |
20070004107 | Lee et al. | Jan 2007 | A1 |
20070040222 | Van Camp et al. | Feb 2007 | A1 |
20070117326 | Tan et al. | May 2007 | A1 |
20070158790 | Rao | Jul 2007 | A1 |
20070212861 | Chidambarrao et al. | Sep 2007 | A1 |
20070238253 | Tucker | Oct 2007 | A1 |
20080001171 | Tezuka et al. | Jan 2008 | A1 |
20080067589 | Ito et al. | Mar 2008 | A1 |
20080108208 | Arevalo et al. | May 2008 | A1 |
20080138953 | Challa et al. | Jun 2008 | A1 |
20080143423 | Komatsu et al. | Jun 2008 | A1 |
20080169493 | Lee et al. | Jul 2008 | A1 |
20080169516 | Chung | Jul 2008 | A1 |
20080197439 | Goerlach et al. | Aug 2008 | A1 |
20080227250 | Ranade et al. | Sep 2008 | A1 |
20080237661 | Ranade et al. | Oct 2008 | A1 |
20080258198 | Bojarczuk et al. | Oct 2008 | A1 |
20080272409 | Sonkusale et al. | Nov 2008 | A1 |
20090003105 | Itoh et al. | Jan 2009 | A1 |
20090057746 | Sugll et al. | Mar 2009 | A1 |
20090057762 | Bangsaruntip et al. | Mar 2009 | A1 |
20090108350 | Cai et al. | Apr 2009 | A1 |
20090121298 | Furukawa et al. | May 2009 | A1 |
20090134468 | Tsuchiya et al. | May 2009 | A1 |
20090224319 | Kohli | Sep 2009 | A1 |
20090302388 | Cai et al. | Dec 2009 | A1 |
20090309140 | Khamankar et al. | Dec 2009 | A1 |
20090311837 | Kapoor | Dec 2009 | A1 |
20090321849 | Miyamura et al. | Dec 2009 | A1 |
20100012988 | Yang et al. | Jan 2010 | A1 |
20100038724 | Anderson et al. | Feb 2010 | A1 |
20100055886 | Izumida et al. | Mar 2010 | A1 |
20100100856 | Mittal | Apr 2010 | A1 |
20100148153 | Hudait et al. | Jun 2010 | A1 |
20100149854 | Vora | Jun 2010 | A1 |
20100187641 | Zhu et al. | Jul 2010 | A1 |
20100207182 | Paschal | Aug 2010 | A1 |
20100270600 | Inukai et al. | Oct 2010 | A1 |
20110059588 | Kang | Mar 2011 | A1 |
20110073961 | Dennard et al. | Mar 2011 | A1 |
20110074498 | Thompson et al. | Mar 2011 | A1 |
20110079860 | Verhulst | Apr 2011 | A1 |
20110079861 | Shifren et al. | Apr 2011 | A1 |
20110095811 | Chi et al. | Apr 2011 | A1 |
20110147828 | Murthy et al. | Jun 2011 | A1 |
20110148945 | Matsuda | Jun 2011 | A1 |
20110169082 | Zhu et al. | Jul 2011 | A1 |
20110175170 | Wang et al. | Jul 2011 | A1 |
20110180880 | Chudzik et al. | Jul 2011 | A1 |
20110193164 | Zhu | Aug 2011 | A1 |
20110212590 | Wu et al. | Sep 2011 | A1 |
20110230039 | Mowry et al. | Sep 2011 | A1 |
20110242921 | Tran et al. | Oct 2011 | A1 |
20110248352 | Shifren et al. | Oct 2011 | A1 |
20110294278 | Eguchi et al. | Dec 2011 | A1 |
20110309447 | Arghavani et al. | Dec 2011 | A1 |
20120021594 | Gurtej et al. | Jan 2012 | A1 |
20120034745 | Colombeau et al. | Feb 2012 | A1 |
20120056275 | Cai et al. | Mar 2012 | A1 |
20120065920 | Nagumo et al. | Mar 2012 | A1 |
20120108050 | Chen et al. | May 2012 | A1 |
20120132998 | Kwon et al. | May 2012 | A1 |
20120138953 | Cai et al. | Jun 2012 | A1 |
20120146155 | Hoentschel et al. | Jun 2012 | A1 |
20120167025 | Gillespie et al. | Jun 2012 | A1 |
20120187491 | Zhu et al. | Jul 2012 | A1 |
20120190177 | Kim et al. | Jul 2012 | A1 |
20120223363 | Kronholz et al. | Sep 2012 | A1 |
Number | Date | Country |
---|---|---|
0274278 | Jul 1988 | EP |
0312237 | Apr 1989 | EP |
0531621 | Mar 1993 | EP |
0683515 | Nov 1995 | EP |
0889502 | Jan 1999 | EP |
1450394 | Aug 2004 | EP |
59193066 | Jan 1984 | JP |
4186774 | Mar 1992 | JP |
8288508 | Jan 1996 | JP |
8153873 | Jun 1996 | JP |
2004087671 | Mar 2004 | JP |
10-0794094 | Jul 2003 | KR |
1020050099328 | Oct 2005 | KR |
2011062788 | May 2011 | WO |
Entry |
---|
Bibliographic information and English Translation of Abstract for KR1020050099328 submitted herewith. |
English Translation of JP 8153873 Submitted herewith. |
Abiko, H et al., “A Channel Engineering Combined with Channel Epitaxy Optimization and TED Suppression for 0.15 μm n-n Gate CMOS Technology”, 1995 Symposium on VLSI Technology Digest of Technical Papers, 1995, pp. 23-24. |
Chau, R et al., “A 50nm Depleted-Substrate CMOS Transistor (DST)”, Electron Device Meeting 2001, IEDM Technical Digest, IEEE International, 2001, pp. 29.1.1-29.1.4. |
Ducroquet, F et al. “Fully Depleted Silicon-On-Insulator nMOSFETs with Tensile Strained High Carbon Content Si1-yCy Channel”, 2006, ECS 210th Meeting, Abstract 1033. |
Ernst, T et al., “Nanoscaled MOSFET Transistors on Strained Si, SiGe, Ge Layers: Some Integration and Electrical Properties Features”, 2006, ECS Trans. 2006, vol. 3, Issue 7, pp. 947-961. |
Goesele, U et al., Diffusion Engineering by Carbon in Silicon, 2000, Mat. Res. Soc. Symp. vol. 610. |
Hokazono, A et al., “Steep Channel & Halo Profiles Utilizing Boron-Diffusion-Barrier Layers (Si:C) for 32 nm Node and Beyond”, 2008, 2008 Symposium on VLSI Technology Digest of Technical Papers, pp. 112-113. |
Hokazono, A et al., “Steep Channel Profiles in n/pMOS Controlled by Boron-Doped Si:C Layers for Continual Bulk-CMOS Scaling”, 2009, IEDM09-676 Symposium, pp. 29.1.1-29.1.4. |
Holland, OW and Thomas, DK “A Method to Improve Activation of Implanted Dopants in SiC”, 2001, Oak Ridge National Laboratory, Oak Ridge, TN. |
Kotaki, H., et al., “Novel Bulk Dynamic Threshold Voltage MOSFET (B-DTMOS) with Advanced Isolation (SITOS) and Gate to Shallow-Well Contact (SSS-C) Processes for Ultra Low Power Dual Gate CMOS”, 1996, IEDM 96, pp. 459-462. |
Lavéant, P. “Incorporation, Diffusion and Agglomeration of Carbon in Silicon”, 2002, Solid State Phenomena, vols. 82-84, pp. 189-194. |
Noda, K et al., “A 0.1- μm Delta-Doped MOSFET Fabricated with Post-Low-Energy Implanting Selective Epitaxy”, Apr. 1998, IEEE Transactions on Electron Devices, vol. 45, No. 4, pp. 809-814. |
Ohguro, T et al., “An 0.18- μm CMOS for Mixed Digital and Analog Aplications with Zero-Volt-Vth Epitaxial-Channel MOSFET's”, Jul. 1999, IEEE Transactions on Electron Devices, vol. 46, No. 7, pp. 1378-1383. |
Pinacho, R et al., “Carbon in Silicon: Modeling of Diffusion and Clustering Mechanisms”, Aug. 2002, Journal of Applied Physics, vol. 92, No. 3, pp. 1582-1588. |
Robertson, LS et al., “The Effect of Impurities on Diffusion and Activation of Ion Implanted Boron in Silicon”, 2000, Mat. Res. Soc. Symp. vol. 610. |
Scholz, R et al., “Carbon-Induced Undersaturation of Silicon Self-Interstitials”, Jan. 1998, Appl. Phys. Lett. 72(2), pp. 200-202. |
Scholz, RF et al., “The Contribution of Vacancies to Carbon Out-Diffusion in Silicon”, Jan. 1999,Appl. Phys. Lett., vol. 74, No. 3, pp. 392-394. |
Stolk, PA et al., “Physical Mechanisms of Transient Enhanced Dopant Diffusion in Ion-Implanted Silicon”, May 1997, J. Appl. Phys. 81(9), pp. 6031-6050. |
Thompson, S et al., “MOS Scaling: Transistor Challenges for the 21st Century”, 1998, Intel Technology Journal Q3' 1998, pp. 1-19. |
Wann, C. et al., “Channel Profile Optimization and Device Design for Low-Power High-Performance Dynamic-Threshold MOSFET”, 1996, IEDM 96, pp. 113-116. |
Werner, P et al., “Carbon Diffusion in Silicon”, Oct. 1998, Applied Physics Letters, vol. 73, No. 17, pp. 2465-2467. |
Yan, Ran-Hong et al., “Scaling the Si MOSFET: From Bulk to SOI to Bulk”, Jul. 1992, IEEE Transactions on Electron Devices, vol. 39, No. 7. |
Dreslinkski, Ronald G. et al., “Near-Threshold Computing: Reclaiming Moore's Law Through Energy Efficient Integrated Circuits”, Feb. 2010, Proceedings of the IEEE, vol. 98, Issue 2, IEEE. |
Komaragiri, R. et al., “Depletion-Free Poly Gate Electrode Architecture for Sub 100 Nanometer CMOS Devices with High-K Gate Dielectrics”, IEEE IEDM Tech Dig., San Francisco CA, 833-836, Dec. 13-15, 2004. |
Samsudin, K et al., Integrating Intrinsic Parameter Fluctuation Description into BSIMSOI to Forecast sub-15 nm UTB SOI based 6T SRAM Operation, Solid-State Electronics (50), pp. 86-93. |
Wong, H et al., “Nanoscale CMOS”, Proceedings of the IEEE, Vo. 87, No. 4, pp. 537-570. |
Machine Translation of KR 10-0794094 Submitted herewith. |
Banerjee et al., “Compensating Non-Optical Effects using Electrically-Driven Optical Proximity Correction”, Proc. of SPIE, vol. 7275, 2009. |
Cheng et al., “Extremely Thin SOI (ETSOI) CMOS with Record Low Variability for Low Power System-on-Chip Applications”, IEDM 2009, Dec. 2009. |
Cheng et al., “Fully Depleted Extremely Thin SOI Technology Fabricated by a Novel Integration Scheme Featuring Implant-Free, Zero-Silicon-Loss, and Faceted Raised Source/Drain”, 2009 Symposium on VLSI Technology Digest of Technical Papers, 2009. |
Drennan et al., “Implications of Proximity Effects for Analog Design”, Custom Integrated Circuits Conference, 2006, CICC '06, IEEE, Sep. 10-13, 2006, pp. 169-176. |
Hook et al., “Lateral Ion Implant Straggle and Mask Proximity Effect”, IEEE Transactions on Electron Devices, vol. 50, No. 9, Sep. 2003. |
Hori et al., “A 0.1 um CMOS with a Step Channel Profile Formed by Ultra High Vacuum CVD and In-Situ Doped Ions”, IEDM 1993, May 12, 1993. |
Matsuhashi et al., “High-Performance Double-Layer Epitaxial-Channel PMOSFET Compatible with a Single Gate CMOSFET”, 1996 Symposium on VLSI Technology Digest of Technical Papers, 1996. |
Shao et al., “Boron diffusion in silicon: the anomalies and control by point defect engineering”, Materials Science and Engineering R 42 (2003), Nov. 2003, pp. 65-114. |
Sheu et al., “Modeling the Well-Edge Proximity Effect in Highly Scaled MOSFETs”, IEEE Transactions on Electron Devices, vol. 53, No. 11, Nov. 2006, pp. 2792-2798. |
Number | Date | Country | |
---|---|---|---|
20160026207 A1 | Jan 2016 | US |