CA116:75710, Murad, S. et al, Arch. Biochem. Biophys., 1992, 292(1), 234-8, abstract.* |
Medline AN 96084808, Karmeli, F. et al, GUT, Sep. 1995, 37(3), 386-93, abstract.* |
C.J. Bulpitt et al., Vitamin C and blood pressure, J. Hypertens. 8(12):1071-5 (1990). |
D. Giugliano et al., Diabetes Mellitus, hypertension, and cardiovascular disease: which role for oxidative stress?, Metabolism 44(3):363-8 (1995). |
S. Grunfeld et al., Role of Superoxide in the depressed nitric oxide production by the endothelium of genetically hypertensive rats, Hypertension 26(6 Pt 1): 854-7 (1995). |
R.J. Gryglewski et al., Superoxide anion is involved in the breakdown of endothelium—derived vascular relaxing factor, Nature 320(6061): 454-6 (1986). |
D.G. Harrison et al., Physiologic consequences of increased vascular oxidant stresses in hypercholesterolemia and atherosclerosis: implications for impaired vasomotion, Am J. Cardiol 75(6): 75B-81B (1995). |
V.V. Khramtsov et al., In vitro and in vivo studies of the derivatives of 1,2-diazetine and nitronylnitroxide as donors and acceptors of nitric oxide. Biokhimiya, 61(10): 1731-1742 (1996). abstract. |
K.V. Kumar et al. Are free radicals involved in the pathobiology of human essential hypertension?, Ree Radic Res Commun. 19(1): 59-66 (1993). |
J. M. McCord, Oxygen-derived free radicals in postischemic tissue injury, N. Engl. J. Med. 312(3): 159-63 (1985). |
Y. Miyamoto et al., “Potentiation of nitric oxide-mediated vasorelaxation by xanthine oxidase inhibitors,” Proc. Soc. Exp. Biol. Med. 312(4): 366-373 (1996). |
K. Nakazono et al., Does superoxide underlie the pathogenesis of hypertension? Proc. Natl. Acad. Sci. USA 88: 10045-10048 (1991). |
G.M. Rubanyi et al., Superoxide anions and hyperoxia inactivate endothelium-derived relaxing factor. Am. J. Physiol. 250: H822-H827 (1986). |
C.G. Schnackenberg et al. Normalization of blood pressure and renal vascular resistance in SHR with a membrane-permeable superoxide dismutase mimetic. Hypertension, 32(1): 59-64 (1998). |
C.G. Schnackenberg et al., Long-term tempol administration attenuates the hypertension and production of 8-iso prostaglandin F2a in SHR. Hypertension 32(3): 622 (1998). |
H. Suzuki et al., In vivo evidence for microvascular oxidative stress in spontaneously hypertensive rats. Hydroethidine microfluorography. Hypertension 25(5): 1083-1089 (1995). |
M.R. Tschudi et al., Direct in situ measurement of nitric oxide in mesenteric arteries. Increased decomposition by superoxide in hypertension. Hypertension 27(1): 32-35 (1996). |
W.Y. Tse et al., Antioxidant status in controlled and uncontrolled hypertension and its relationship to endothelial damage. J. Hum. Hypertens. 89: 843-849 (1994). |
M. Yoshioka et al., Effects of ascorbic acid on blood pressure and ascorbic acid metabolism in spontaneously hypertensive rats (SH Rats). Internat. J. Vit. Nutr. Res. 55: 301-307 (1985). |
Tal, Neuroreport (1996) 7(8) 1382-84. |
Karmeli et al. (1996) GUT 38(6) 826-31. |
Monte et al. (1996) Free Radical Biol. Med. 21(4) 463-70. |
Wilcox (2000) New Endothelium-Derived Metabolites of Arachidonic Acid that Mediate Vasodilation. Lewis K. Dahl Memorial Lecture, American Heart Association. |